Apoptosis
Apoptosis, also known as programmed cell death, is rigorously controlled process of cell death that leads to phagocytosis of unwanted cell. It is triggered after sufficient cellular damage and activated through extrinsic or intrinsic pathways. The intrinsic pathway is mainly occurs via release of cytochrome c from the mitochondria and regulates mitochondrial outer membrane permeabilization by Bcl-2 family proteins. The extrinsic pathway is induced by ligand binding to death receptor, such as Fas, TNFαR, DR3, DR4, and DR5. Caspases then cleave target proteins and nuclear lamins to promote DNA degradation, resulting apoptotic cells undergo phagocytosis. In addition, p53 has the ability to activate intrinsic and extrinsic pathways of apoptosis by inducing transcription of several proteins like Puma, Bid, Bax, TRAIL-R2, and CD95.
Some Inhibitors of apoptosis proteins (IAPs), such as XIAP/BIRC4 and Bruce/BIRC6, can block casapse activity through direct binding, while other IAPs, such as cIAP1/BIRC2, cIAP2/BIRC3, act as ubiquitin ligases that target caspases for ubiquitin-mediated degradation. Apoptosis is essential for growth, development and aging in multicellular organisms. Any alterations or abnormalities occurring in apoptotic processes contribute to development of human diseases, including cancer.
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A9957 Myristoleic acidSummary: A monounsaturated fatty acid that induces apoptosis and necrosis of human prostate cells -
C8445 CWI1-2 hydrochloride -
C8447 TNF-α-IN-1 -
C8452 Anticancer agent 43 -
C8466 BAY 1892005 -
C8493 SIRT6 activator 12q -
C8496 Anti-melanoma agent 1 -
C8522 Apoptotic agent-3 -
C8547 BAI1 hydrochloride -
BA2719 DT2216Summary: DT2216 is an effective, selective (family member) degrader of a class.


